Search results for "Viral genomes"

showing 9 items of 9 documents

2018

In many viral infections, a large number of different genetic variants can coexist within a host, leading to more virulent infections that are better able to evolve antiviral resistance and adapt to new hosts. But how is this diversity maintained? Why do faster-growing variants not outcompete slower-growing variants, and erode this diversity? One hypothesis is if there are mutually beneficial interactions between variants, with host cells infected by multiple different viral genomes producing more, or more effective, virions. We modelled this hypothesis with both mathematical models and simulations, and found that moderate levels of beneficial coinfection can maintain high levels of coexist…

0301 basic medicineGeneticsHost (biology)Genetic variantsAntiviral resistanceVirulenceBiologymedicine.diseaseMicrobiology03 medical and health sciencesMultipartite030104 developmental biologyViral genomesVirologyCoinfectionmedicineDiversity (business)Virus Evolution
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Collective Infectious Units in Viruses

2017

Increasing evidence indicates that viruses do not simply propagate as independent virions among cells, organs, and hosts. Instead, viral spread is often mediated by structures that simultaneously transport groups of viral genomes, such as polyploid virions, aggregates of virions, virion-containing proteinaceous structures, secreted lipid vesicles, and virus-induced cell-cell contacts. These structures increase the multiplicity of infection, independently of viral population density and transmission bottlenecks. Collective infectious units may contribute to the maintenance of viral genetic diversity, and could have implications for the evolution of social-like virus-virus interactions. These…

0301 basic medicineMicrobiology (medical)virusesBiologyMicrobiologyArticle03 medical and health sciencesMultiplicity of infectionImmunityVirologyAnimalsGeneticsGenetic diversityVirionGenetic VariationBiological EvolutionVirologyMicrovesiclesComplementation030104 developmental biologyInfectious DiseasesVirus DiseasesViral genomesViral spreadLipid vesicleBaculoviridaeTrends in Microbiology
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2019

Viruses frequently spread among cells or hosts in groups, with multiple viral genomes inside the same infectious unit. These collective infectious units can consist of multiple viral genomes inside the same virion, or multiple virions inside a larger structure such as a vesicle. Collective infectious units deliver multiple viral genomes to the same cell simultaneously, which can have important implications for viral pathogenesis, antiviral resistance, and social evolution. However, little is known about why some viruses transmit in collective infectious units, whereas others do not. We used a simple evolutionary approach to model the potential costs and benefits of transmitting in a collect…

0303 health sciencesCancer Research030306 microbiologyvirusesViral pathogenesisAntiviral resistanceBiologyVirologyGenome03 medical and health sciencesInfectious DiseasesMultiplicity of infectionViral replicationViral genomesVirologyViral evolution030304 developmental biologyVirus Research
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Virus entéricos humanos en alimentos: detección y métodos de inactivación

2020

[ES] Los principales patógenos víricos que podemos adquirir ingiriendo alimentos contaminados son los norovirus, el virus de la hepatitis A y el virus de la hepatitis E que se propagan principalmente a través de la vía fecal oral. En los últimos años, la incidencia de brotes de transmisión alimentaria causados por estos patógenos ha experimentado un aumento considerable, en parte debido al comercio globalizado y a los cambios en los hábitos de consumo. Las matrices alimentarias que mayor riesgo representan para el consumidor son los moluscos bivalvos, vegetales de IV gama, frutas tipo baya y platos listos para comer. Actualmente las técnicas moleculares son las más habituales para la detecc…

Cultural StudiesSociology and Political ScienceFood industryViral inactivationmolecular methodsFoodborne virusesBiologymedicine.disease_causeGeneral WorksFood safety03 medical and health sciencesseguridad alimentariaAmedicineenvases virucidas030304 developmental biologyinactivación víricaInfectivityviral inactivationmetagenomicscompuestos virucidas0303 health sciencesantiviral packaging030306 microbiologybusiness.industryvirus entéricosGeneral Arts and Humanitiesdigestive oral and skin physiologyfoodborne virusesFoodborne outbreakHepatitis AFood safetymedicine.diseaseVirologyantiviral compoundsVirusMolecular methodsfood safetymétodos molecularesAliments ContaminacióViral genomesAntiviral packagingmetagenómicaNorovirusAntiviral compoundsMetagenomicsbusinessContaminated foodArbor
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Why are viral genomes so fragile? The bottleneck hypothesis

2021

If they undergo new mutations at each replication cycle, why are RNA viral genomes so fragile, with most mutations being either strongly deleterious or lethal? Here we provide theoretical and numerical evidence for the hypothesis that genetic fragility is partly an evolutionary response to the multiple population bottlenecks experienced by viral populations at various stages of their life cycles. Modelling within-host viral populations as multi-type branching processes, we show that mutational fragility lowers the rate at which Muller’s ratchet clicks and increases the survival probability through multiple bottlenecks. In the context of a susceptible-exposed-infectious-recovered epidemiolog…

Evolutionary GeneticsRNA virusesMutation rateEpidemiologyExtinct GenomesMedicine and Health SciencesBiology (General)Genetics0303 health sciencesEvolutionary epidemiologyEcologyMicrobial MutationGenomicsDeletion MutationComputational Theory and MathematicsViral genomesGenetic EpidemiologyModeling and SimulationViral evolutionPopulation bottlenecksVirusesRNA ViralResearch ArticleQH301-705.5Genomics[SDV.CAN]Life Sciences [q-bio]/CancerContext (language use)Genome ViralBiologyMicrobiologyGenomic InstabilityViral EvolutionBottleneckEvolution Molecular03 medical and health sciencesCellular and Molecular NeuroscienceSurvival probabilityVirologyGeneticsFragilityMolecular BiologyEcology Evolution Behavior and Systematics030304 developmental biologyEvolutionary BiologyModels Genetic030306 microbiologyOrganismsComputational BiologyBiology and Life SciencesRNAVirus evolutionOrganismal EvolutionGenetic architecture[MATH.MATH-PR]Mathematics [math]/Probability [math.PR]Population bottleneckViral replicationMutationMicrobial Evolution
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Inverted Repeats in Viral Genomes

2004

We investigate 738 complete genomes of viruses to detect the presence of short inverted repeats. The number of inverted repeats found is compared with the prediction obtained for a Bernoullian and for a Markovian control model. We find as a statistical regularity that the number of observed inverted repeats is often greater than the one expected in terms of a Bernoullian or Markovian model in several of the viruses and in almost all those with a genome longer than 30,000 bp.

Genomics (q-bio.GN)Statistical Mechanics (cond-mat.stat-mech)Complex systemInverted repeatGeneral Mathematicsviral genomeGeneral Physics and AstronomyFOS: Physical sciencesComputational biologyBiologyGenomeQuantitative Biology - Quantitative MethodsSettore FIS/07 - Fisica Applicata(Beni Culturali Ambientali Biol.e Medicin)stochastic processeViral genomesFOS: Biological sciencessecondary RNA struc- tureQuantitative Biology - GenomicsQuantitative Methods (q-bio.QM)Condensed Matter - Statistical MechanicsDNA probabilistic models
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Statistical properties of thermodynamically predicted RNA secondary structures in viral genomes

2008

By performing a comprehensive study on 1832 segments of 1212 complete genomes of viruses, we show that in viral genomes the hairpin structures of thermodynamically predicted RNA secondary structures are more abundant than expected under a simple random null hypothesis. The detected hairpin structures of RNA secondary structures are present both in coding and in noncoding regions for the four groups of viruses categorized as dsDNA, dsRNA, ssDNA and ssRNA. For all groups hairpin structures of RNA secondary structures are detected more frequently than expected for a random null hypothesis in noncoding rather than in coding regions. However, potential RNA secondary structures are also present i…

Genomics (q-bio.GN)inverted repeatbioinformaticRNAstatistical physicsComputational biologyBiologyCondensed Matter PhysicsGenomeQuantitative Biology - Quantitative MethodsElectronic Optical and Magnetic MaterialsRNA silencingViral genomesFOS: Biological sciencesCoding regionQuantitative Biology - GenomicsQuantitative Methods (q-bio.QM)
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The distribution of fitness effects caused by single-nucleotide substitutions in an RNA virus.

2004

6 pages, 3 figures.-- PMID: 15159545 [PubMed].-- PMCID: PMC420405.-- Supporting information (Table 3: Relevant information about each single-nucleotide substation mutant created) available at: http://www.pnas.org/content/101/22/8396/suppl/DC1

PopulationMutantMutagenesis (molecular biology technique)Evolutionary biologyVesicular stomatitis Indiana virusSingle-nucleotide substitutionsGenetic variationAnimalsPoint MutationMutational fitness effectseducationGeneticseducation.field_of_studyMultidisciplinarybiologyPoint mutationRNAGenetic VariationRNA virusRNA viral genomesBiological Sciencesbiology.organism_classificationBiological EvolutionGenetics PopulationVesicular stomatitis virusMutagenesis Site-DirectedProceedings of the National Academy of Sciences of the United States of America
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The evolution of collective infectious units in viruses

2019

Viruses frequently spread among cells or hosts in groups, with multiple viral genomes inside the same infectious unit. These collective infectious units can consist of multiple viral genomes inside the same virion, or multiple virions inside a larger structure such as a vesicle. Collective infectious units deliver multiple viral genomes to the same cell simultaneously, which can have important implications for viral pathogenesis, antiviral resistance, and social evolution. However, little is known about why some viruses transmit in collective infectious units, whereas others do not. We used a simple evolutionary approach to model the potential costs and benefits of transmitting in a collect…

Viral pathogenesisviruseseducationGenome ViralBiologyVirus ReplicationGenomebehavioral disciplines and activitiesArticleEvolution Molecular03 medical and health sciences0302 clinical medicine030304 developmental biology0303 health sciencesVirus AssemblyAntiviral resistanceVirionDefective VirusesModels TheoreticalVirologyViral replicationViral genomesVirus Diseasespopulation characteristicsRNA Viral030217 neurology & neurosurgery
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